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In untreated galactosemic patients, isoform patterns of serum transferrin, lysosomal enzymes b-hexosaminidase and a-fucosidase and follicle stimulating hormone are abnormal, due to the increase of relatively neutral isoforms corresponding to less sialylated carbohydrate structures [1-2]
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4i) which showed formation of CAM-S-CAM-TPP at a similar concentration to GSS-CAM-TPP (Fig
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