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Calcium entry through the compromised plasma membrane has been shown to activate proteases (calpain, calcineurin), resulting in neurofilament degradation, disruption of axonal transport, and functional failure (Schlaepfer, 1974, 1987)
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[17] The peptide appeared to help preserve the normal structure of the eye while also supporting the bioelectric activity of the retina, which is essential for vision signaling
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