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[DOI] [PMC free article] [PubMed] [Google Scholar] 141.Demir M., Altindag F
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This is supported by postmortem brain analyses showing increased levels of 4-hydroxyl-2-nonenal (HNE), a by-product of lipid peroxidation [8, 9], carbonyl modifications of soluble proteins [10], and DNA and RNA oxidation products 8-hydroxy-deoxyguanosine and 8-hydroxy-guanosine [11, 12]
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