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Until technologies for SOD1 metalloform quantification are refined, and expression systems are made to more accurately replicate human physiology, it remains unclear whether altered SOD1 metalation in common models of mutant and wild‐type SOD1 misfolding is a product of destabilizing factors, expression systems, or isolation and quantification procedures, and thus whether SOD1 mismetalation possesses any relevance to human health or disease

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