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These results suggest that HAdV-C5 does not activate NF-κB in a manner similar to TNFα and it can be assumed that HAdV-C5 encodes for a protein that counteracts NF-κB activity

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Recent evidence indicates that AD brains exhibit canonical ferroptotic pathology, which drives mitochondrial cardiolipin peroxidation, causes ultrastructural damage, impairs ATP synthesis, and ultimately triggers irreversible neurodegenerative decline ( Figure 3 ) (186)

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