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Collectively, our data indicate multiple complement-inhibitory activities of these molecules, redox-sensitive cysteine motifs (ChiC, ChiE), and a putative catalytic triad (ChiE)

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As a consequence, the invasion of gram negative enterobacteria initiate the inflammatory cascade wherein Toll like receptor-4 (TLR-4)-induced proinflammatory cytokine upsurge along with NLRP3 inflammosome activation attract the neutrophils eventually leading to oxidative burst, chronic inflammation, apoptosis and ultimately intestinal ulceration [140]