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TB-500’s key mechanisms include: Actin sequestration: By binding G-actin (monomeric actin), TB-500 keeps it available for cell migration and wound repair rather than polymerizing prematurely into structural filaments
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Antacids to reduce gastric acid secretion If a patient is not eating then a feeding tube (o-tube) may be placed to facilitate adequate nutrition and stimulate pancreatic secretion (unless the patient is experiencing vomiting)
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