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This multitude (Table 1) clearly demonstrates a multimodal muscle axis impact that is able to react depending on the condition and the given agent(s) and the symptoms that are distinctively related to the primary cause of injury within the wider healing concept, where the implementation of the concept of the cytoprotection [1,3,4,5,6,8,9,32,33,34] has appeared as novel point

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Further investigation of the fetal heart indicates that disrupting ROS levels by overexpressing agents such as N-acetylcysteine or the SOD1 gene can effectively reduce ROS levels, consequently decreasing the incidence of CHDs (Moazzen et al., 2014