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This hyperactive Notch signaling, previously identified as a cause of delayed neurogenesis (Levy et al., 2002), has been demonstrated to be a mechanism implicated in the disease and suggests a novel mitochondrial-Notch crosstalk previously unrecognized in MELAS
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The thicknesses of 3% alginate or 5% gelatin-methacryloyl hydrogel encapsulation optimally preserved mouse testicular tissues by inhibiting ice crystal formation, minimizing basement membrane contraction, improving cell morphology, and augmenting mitochondrial activity (14)
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